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Given the varied mechanisms by which antiarrhythmic drugs modify cardiac action potentials and conduction velocities to treat arrhythmias, how do these mechanisms align with the selection of specific antiarrhythmic medications for conditions such as atrial flutter, atrial fibrillation, and AV block, considering both the intended therapeutic outcomes and the potential for adverse effects

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Another possible explanation for the mixed results is that oxytocins effect on social behavior is limited when the treatment context lacks social cues