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According to the classic two-hits model, the increased insulin resistance, secondary to metabolic syndrome, determines an excessive lipid accumulation in healthy hepatocytes (first hit), mainly through an increased mobilization of free fatty acids from visceral adipose tissue to the liver

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And it does not eliminate the need for the one thing that protects patients more than any regulatory change: a physician who understands their biology, orders the appropriate labs, determines clinical indication, and monitors their response

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I don't agree that's the case