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However, the enhanced density of mitochondria and upregulated mitochondrial complex IV activity in MS lesion sites are also observed to be associated with increased mitochondrial stress protein mtHSP70, suggesting that enhanced density of mitochondria in MS lesions might also contribute to the formation of free radicals and subsequent tissue damage (Witte et al., 2009)

In one early trial, no significant changes in [I]FP-CIT SPECT uptake were observed over 12 months, likely due to the advanced disease stage of participants [54]

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