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163 Notably, remifentanil self-administration was comparable to or increased in mice lacking central nervous system GLP-1 receptors relative to wild-type controls
Some C1 area (TH-positive and -negative) neurons were confirmed to be bulbospinal presympathetic neurons because they were antidromically activated by ipsilateral IML stimulation, which is basically consistent with the results of previous studies [26, 32,33,34]
It is rare, but the immune-modulatory effects of weight loss and hormonal changes could theoretically trigger autoimmune responses in susceptible individuals
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