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The distinction between adaptive and maladaptive muscle loss is central to understanding the quality, not just the quantity, of BW loss achieved with GLP-1 RA-based therapies [37, 38]
In the present study, we investigated whether activation of GLP-1R prevented allodynia and inhibited microglial morphological changes and inflammation in the TNC using a chronic nitroglycerin (NTG) injection-stimulated mouse model and the selective GLP-1R agonist liraglutide
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This boosts GLP-1 secretion in the gut, which reduces blood sugar, appetite and inflammation levels
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