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These findings help to explain why leptin replacement does not completely reverse the neuroendocrine adaptations induced by weight loss 1, 3, 5 , since both GH and leptin play a role informing the brain about energy deficiency
*Correspondence: Ze Wang, [email protected] Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers
AS, GL, and SCS secured funding
The measured position is that the pathway is important and GHK-Cu is pleiotropic, so overlap is likely but a clean, primary demonstration that GHK-Cu drives Wnt-dependent anagen in human follicles is not something the record firmly establishes
From this follows an important methodological point
The hydroxyl groups of CD allow structural modifications yielding hydrophobic derivatives, namely acylated and ethylated cyclodextrins, that may be used to achieve a sustained release of the drugs
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