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Taken together, a negative feedback effect of GSSSG on LPS-induced activation of the NF-B signaling pathway and cytokine production may explain the mechanism underlying the GSSSG-mediated inhibition of microglial proliferation

RO exhibit potent oxidative activity, abstracting hydrogen atoms and reacting with biomolecules, thereby causing membrane damage and functional impairment, as well as modulating protein functionality and genomic stability [67]

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