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Interestingly, supplementation with mitochondria-targeted TEMPO (mitoTEMPO), which accumulates in active mitochondria where the reduction of its active nitroxide to hydroxylamine is mainly mediated by ubiquinol 35 , also significantly rescued survival upon combined LDHB silencing and RSL3-treatment (Fig

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[2] [3] The distinction between expected pharmacologic gastric slowing and pathologic gastroparesis requires careful evaluation of both incidence data and clinical presentation

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However, translating mouse fat metabolism data to human outcomes requires substantial caution

It represents a mean difference of 3.1 1.0% between the two groups